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Two studies have evaluated the effect of antibiotics on the rate of expansion of abdominal aortic aneurysms. Activity of gemifloxacin and other new quinolones against Chlamydia pneumoniae: a review. Kaufman M, Gaydos CA, Sriram S et al.
Chlamydia pneumoniae infection of the central nervous system and multiple sclerosis. Roblin PM, Kutlin A, Reznik T, Hammerschlag MR. Its etiology is complex, involving interactions between genetic susceptibility, exposure to allergens and external aggravating factors such as smoking, air pollution and respiratory tract infections. The results of in vitro testing indicate that, unlike C.
Antimicrobial susceptibility and therapy of infections caused by Chlamydia pneumoniae. Microbiologic efficacy of azithromycin and susceptibilities to azithromycin of isolates of Chlamydia pneumoniae from adults and children with community-acquired pneumonia. Attention has been focused on investigating hypothetical additional risk factors and on a fuller understanding of the development of atherosclerosis, the main pathologic process involved in coronary heart disease.
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Block S, Hedrick J, Hammerschlag MR, Cassell GH, Craft JC. Available data on the susceptibility of C. Chlamydia, and were thought to represent strains of C. In vitro activities of azithromycin, clarithromycin, erythromycin, and tetracycline against 13 strains of Chlamydia pneumoniae. The vast majority of studies were concordant in finding an association between atypical bacterial infection and asthma exacerbations.
An MRI study of Chlamydia pneumoniae infection in Italian multiple sclerosis patients. An interesting review on the issue of innate immunity and vaccines in C. Kern JM, Maas V, Maas M. Grayston JT, Kuo CC, Wang SP, Altman J. Welsh L, Gaydos C, Quinn TC. What’s New West, SK et al.
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Pulmonary rales, ronchi or signs of pulmonary consolidation are sometimes found. Chlamydia pneumoniae-induced pathological signaling in the vasculature. No portion of this site may be reproduced. A summary of the results of antimicrobial susceptibility testing from previous published reports is presented in Table 1. Recovery of Chlamydia pneumoniae in six patients with otitis media and effusion.
Grayston JT, Campbell LA, Kuo CC, et al. Role of persistent infection in the control and severity of asthma: focus on Chlamydia pneumoniae. Studies that have used PCR to detect C. In addition, as previously described, persistence of the organism, as documented by detection by isolation or PCR of nasopharyngeal swab specimens, has been documented after treatment despite clinical improvement.
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Balin BJ, Scott Little C, Hammond CJ, Appelet DM, Whittum-Hudson JA, Gerard HC, Hudson AP. Mosorin M, Juvonen J, Biancari F, Satta J, Surcel HM, Leinonen M, Saikku P, Juvonen T. A recent study demonstrated that brain tissue samples from a high proportion of patients with AD are PCR-positive for C.
The Effect of Telithromycin in Acute Exacerbations of Asthma: the TELICAST Study. Emended description of the order Chlamydiales, proposal of Parachlamydiaceae fam. Lack of detectable Chlamydia pneumoniae in brain lesions of patients with multiple sclerosis.
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Lipsky BA, Tack KJ, Wang SP, Kuo CC, Grayston JT. Airenne S, Surcel HM, Tuukkanen J, Leinonen M, Saikku P. Fan T, Lu H, Hu H et al. Lumbosacral meningoradiculitis associated with Chlamydia pneumoniae infection. Blasi F, Aliberti S, Allegra L, et al. Dowell SF, Peeling RW, Boman J et al.
Ke Z, Lu F, Roblin P et al. Qualitative and quantitative detection of Chlamydophila pneumonia DNA in cerebrospinal fluid from multiple sclerosis patients and controls. Microbiologic efficacy of moxifloxacin for the treatment of community-acquired pneumonia due to Chlamydia pneumoniae. Summary of The Results of In Vitro Susceptibility Testing of C.
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Gupta S, Leatham EW, Carrington D, Mendall MA, Kaski JC, Camm AJ. The authors conclude that, considering present evidence, C. Jones RB, Van Der Pol B, Martin DH, Shepard MK.
Marrie TJ, Harczy M, Mann OE, Landymore RW, Raza A, Wang SP, Grayston JT. Atypical pathogen infection in adults with acute exacerbation of bronchial asthma. Chlamydia pneumoniae inhibits apoptosis in human epithelial and monocyte cell lines. In-house nucleic acid amplification assays in research: how much quality control is needed before one can rely upon the results? It has been successfully employed with respiratory specimens, lung and vascular biopsy specimens, and blood.
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MICs on first passage but relatively low MICs on second passage. It has been suggested that there may be an association between acute asthma exacerbations and infection with or reactivation of the atypical bacterium C. Pneumonia and bronchitis are the most common, while upper respiratory infections, including sinusitis and pharyngitis, may also occur, either in isolation or in conjunction with a lower respiratory infection. If symptoms such as cough or malaise persist after one course of antibiotics, a second course may be useful. Chest X-ray generally reveals small pulmonary infiltrates, sublobar or segmental at presentation. In vitro drug susceptibility of Chlamydia sp.
Most investigators also determine the MIC for second passage by harvesting the cells from duplicate plates run in parallel and disrupting and passing the cells onto new monolayers. These results also indicate that tetracyclines, macrolides, and azalides are active against C. Hammerschlag MR, Chirgwin K, Roblin PM, Gelling M, Dumornay W, Mandel L, Smith P, Schachter J. In patients with COPD persistence of micro-organisms in the respiratory tract may facilitate access of different pathogens to the lower airways, and longstanding infection might trigger what is traditionally described as the vicious circle of chronic bronchitis. Cooper MA, Baldwin D, Matthews RS, Andrews JM, Wise R. Partial characterization of Chlamydia trachomatis isolates resistant to multiple antibiotics.
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Severe asthma exacerbation: role of acute Chlamydophila pneumonia and Mycoplasma pneumoniae infection. Cosentini R, Tarsia P, Canetta C, et al. Chlamydophila pneumoniae induces a sustained airway hyperresponsiveness and inflammation in mice. Chlamydia pneumoniae and vascular disease: an update. The clinical course may vary from mild, self-limiting illnesses to severe forms of pneumonia, particularly in elderly patients, and with coexisting cardiopulmonary diseases.
Nagy A, Kozma GT, Keszei M et al. Huovinen P, Lahtonen R, Ziegler T, Meurman O, Hakkarainen K, Miettinen A, Arstila P, Eskola J, Saikku P. The incubation period of infection due to C. Chlamydia pneumoniae-induced atherosclerosis in a rabbit model? Welti M, Jaton K, Altwegg M, Sahli R, Wenger A, Bille J.
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In these studies, the patients showed improvement of clinic symptoms despite persistent culture positivity. Published data suggest the need for longitudinal observations, and clinical trials with C. Rapid and simple diagnosis of Chlamydophila pneumoniae pneumonia by an immunochromatographic test for detection of immunoglobulin M antibodies. Hahn DL, Dodge RW, Golubjatnikov R. Phagocytes transmit Chlamydia pneumoniae from the lung to the vasculature.
A new respiratory tract pathogen: Chlamydia pneumoniae strain TWAR. Is Chlamydia pneumoniae found in spinal fluid samples from multiple sclerosis patients? Acute respiratory infection due to Chlamydia pneumoniae: current status of diagnostic methods. Michel D, Antoine JC, Pozzetto B, Gaudin OG, Lucht F.
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Evidence for infection with Chlamydia pneumoniae in a subgroup of patients with multiple sclerosis. Esposito S, Bosis S, Faelli N, et al. Presenting symptoms most frequently reported by patients with C. Grimaldi LM, Pincherle A, Martinelli-Boneschi F et al.
Recently, a new method for rapid diagnosis of C. In: The Cochrane Library, Issue 1. Richeldi L, Ferrara G, Fabbri LM, et al. Fever may be often missed if the patient is not seen early in the course of infection. Muhlestein JB, Anderson JL, Hammond EH, Zhao L, Trehan S, Schwobe EP, Carlquist JF. More recently, the results of several clinical trials which have included nasopharyngeal culture positivity as an endpoint have been reported.